Please use this identifier to cite or link to this item: http://hdl.handle.net/20.500.12779/7221
DC FieldValueLanguage
dc.contributor.authorFusi, Fabioen_us
dc.contributor.authorTrezza, Alfonsoen_us
dc.contributor.authorSgaragli, G.en_us
dc.contributor.authorSpiga, Ottaviaen_us
dc.contributor.authorSaponara, S.en_us
dc.contributor.authorBova, S.en_us
dc.date.accessioned2021-03-30T16:10:33Z-
dc.date.available2021-03-30T16:10:33Z-
dc.date.issued2020-
dc.identifier.issn1671-4083en_US
dc.identifier.urihttp://hdl.handle.net/20.500.12779/7221-
dc.description222302en_US
dc.language.isoenen_US
dc.relationNoneen_US
dc.relation.ispartofACTA PHARMACOLOGICA SINICAen_US
dc.subject5-HT; 2; receptor; Ca; V; 1.2 channel; docking simulation; homology modeling; Raynaud’s syndrome; ritanserinen_US
dc.titleRitanserin blocks CaV1.2 channels in rat artery smooth muscles: electrophysiological, functional, and computational studiesen_US
dc.typeArticleen_US
dc.identifier.doi10.1038/s41401-020-0370-1en_US
dc.identifier.pmid32132658en_US
dc.identifier.scopus2-s2.0-85081560951en_US
dc.identifier.isiWOS:000518096600001en_US
dc.relation.volume41en_US
dc.relation.issue9en_US
dc.description.firstpage1158en_US
dc.description.lastpage1166en_US
dc.description.thirdmissionNot applicableen_US
item.cerifentitytypePublications-
item.grantfulltextnone-
item.languageiso639-1en-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.openairetypeArticle-
item.fulltextNo Fulltext-
crisitem.author.orcid0000-0002-5737-8309-
crisitem.author.orcid0000-0002-0263-7107-
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